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Once vision is lost, it is seldom recoverable For this reason the earliest suspicion of cranial arteritis should lead to biopsy of the appropriate scalp artery and the immediate administration of corticosteroids Microscopic examination discloses an intense granulomatous or giant-cell arteritis If biopsy on one side fails to clarify the situation and there are sound clinical reasons for suspecting the diagnosis, the other side should be sampled Arteriography of the external carotid artery branches is probably the most sensitive test but is seldom used; ultrasound examination has recently been introduced to display a dark halo and irregularly thick-.

subject of a monograph by R W Smith in 1849; even at that time, he referred to examples recorded by other writers It was von Recklinghausen, however, who in 1882 gave the de nitive account of its clinical and pathologic features The subsequent studies of the disease by Yakovlev and Guthrie, Lichtenstein, Riccardi, and Martuza and Eldridge, and more recently by Creange et al, and the comprehensive monographs of Crowe and Riccardi and their colleagues are informative references that provide a complete analysis of the clinical, pathologic, and genetic data pertaining to the disease Epidemiology Crowe and associates have calculated the prevalence of the disease to be 30 to 40 per 100,000, with the expectancy of one case in every 2500 to 3300 births.

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Approximately half of their cases had affected relatives, and in all instances the distribution of cases within a family was consistent with an autosomal dominant mode of inheritance The disease has been observed in all races in different parts of the world, and males and females are about equally affected Cause and Pathogenesis The hereditary nature of NF has been appreciated for many years More recently it has been established that NF comprises two distinct disorders, the genes for which are located on different chromosomes Both are inherited in an autosomal dominant pattern with a high degree of penetrance, but half the cases are due to new mutations The classic form of the disease with multiple neuro bromas, described below, is caused by a mutation located near the centromere on chromosome 17 in a gene called neuro bromin (Barker et al).

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The second type, in which the main feature is bilateral acoustic nerve neuromas, described further on, is caused by a gene termed merlin (also called schwannomin) These two forms of NF have been loosely referred to as peripheral and central, respectively, but the terms neuro bromatosis type 1 (NF1) and neuro bromatosis type 2 (NF2) are less confusing (Martuza and Eldridge) and are used in the following discussion The pathogenesis is less obscure now that the genes implicated in both diseases have been identi ed Both involve tumor suppres-.

The theoretical value of the July 18.00 call option after 40 days and a drop in volatility of 10 points is 31.0 or $1,550.00. We initially paid 67.0 or $3,350 and now have $1,550 remaining with no change in market price. The same could be said if the volatility were to gain 10 points over the period. If you add the premium value from a volatility gain against the time value decay it will help offset the loss from time. With the leverage represented in futures contracts, the monetary gain or loss from changes in volatility can be substantial and relatively quick. Historically speaking volatility tends to build slowly and disappear quickly, especially in futures markets. As a call buyer, you need to monitor the volatility of the market carefully when buying in high volatility. Derivative options can also vary in volatility from contract month to contract month, so you must monitor the volatility for the particular underlying contract as well as the front month volatility for changes in trends that could adversely affect your position. The obvious choice for derivative calls is to buy in

The troposphere is the lower part of the atmosphere extending nominally to 50 km above the surface of the planet. The troposphere is composed essentially of electrically neutral particles and for L-Band signals it is nondispersive. The troposphere experiences changes in temperature, pressure, and humidity associated with weather. Because these same variables a ect the density of the air mass along the signal path and the index-of-refraction is a function of air mass density, tropospheric conditions a ect the measured time-of-propagation with the measured value being larger than that the geometric range. Tropospheric delays

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